anti ccnd2 Search Results


gene exp ccnd2 mm00438070 m1  (Thermo Fisher)
98
Thermo Fisher gene exp ccnd2 mm00438070 m1
Gene Exp Ccnd2 Mm00438070 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/gene exp ccnd2 mm00438070 m1/product/Thermo Fisher
Average 98 stars, based on 1 article reviews
gene exp ccnd2 mm00438070 m1 - by Bioz Stars, 2026-03
98/100 stars
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mouse anti ccnd2  (Boster Bio)
90
Boster Bio mouse anti ccnd2
MiR-198 directly bound to the 3′-UTR of <t>CCND2</t> mRNA. ( A ) Bioinformatics analyses showed that 10 potential target genes of miR-198 were predicted by three different databases; ( B ) The two distinct predicted binding sites of miR-198 in the 3′-UTR of CCND2 mRNA were allocated, and the fragments containing either mutated binding site were amplified according to the mature miR-198 sequence; ( C ) In pMIR-REPORT™ vector, CCND2 mRNA 3′-UTRfragment containing either the wild type or the mutated Site 1 was fused downstream the reporter gene. When the vectors were cotransfected with miR-198 mimic or mimic control, and the relative luciferase activity, normalised by β-gal, was significantly suppressed in vector with wild type Site 1 than that with mutated Site 1 (32.80% ± 6.89%); ( D ) In the presence of wild type Site 2, not the mutated one, miR-198 was able to significantly inhibit luciferase activity although to a less extent than wild type Site 1 (55.39% ± 8.48%). (NC, negative control; WT, wide-type; MT, mutated-type. * Compared with NC, p < 0.05).
Mouse Anti Ccnd2, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti ccnd2/product/Boster Bio
Average 90 stars, based on 1 article reviews
mouse anti ccnd2 - by Bioz Stars, 2026-03
90/100 stars
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anti-ccnd2  (MBL Life science)
90
MBL Life science anti-ccnd2
MiR-198 directly bound to the 3′-UTR of <t>CCND2</t> mRNA. ( A ) Bioinformatics analyses showed that 10 potential target genes of miR-198 were predicted by three different databases; ( B ) The two distinct predicted binding sites of miR-198 in the 3′-UTR of CCND2 mRNA were allocated, and the fragments containing either mutated binding site were amplified according to the mature miR-198 sequence; ( C ) In pMIR-REPORT™ vector, CCND2 mRNA 3′-UTRfragment containing either the wild type or the mutated Site 1 was fused downstream the reporter gene. When the vectors were cotransfected with miR-198 mimic or mimic control, and the relative luciferase activity, normalised by β-gal, was significantly suppressed in vector with wild type Site 1 than that with mutated Site 1 (32.80% ± 6.89%); ( D ) In the presence of wild type Site 2, not the mutated one, miR-198 was able to significantly inhibit luciferase activity although to a less extent than wild type Site 1 (55.39% ± 8.48%). (NC, negative control; WT, wide-type; MT, mutated-type. * Compared with NC, p < 0.05).
Anti Ccnd2, supplied by MBL Life science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti-ccnd2/product/MBL Life science
Average 90 stars, based on 1 article reviews
anti-ccnd2 - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier
cyclind2  (Boster Bio)
90
Boster Bio cyclind2
MiR-198 directly bound to the 3′-UTR of <t>CCND2</t> mRNA. ( A ) Bioinformatics analyses showed that 10 potential target genes of miR-198 were predicted by three different databases; ( B ) The two distinct predicted binding sites of miR-198 in the 3′-UTR of CCND2 mRNA were allocated, and the fragments containing either mutated binding site were amplified according to the mature miR-198 sequence; ( C ) In pMIR-REPORT™ vector, CCND2 mRNA 3′-UTRfragment containing either the wild type or the mutated Site 1 was fused downstream the reporter gene. When the vectors were cotransfected with miR-198 mimic or mimic control, and the relative luciferase activity, normalised by β-gal, was significantly suppressed in vector with wild type Site 1 than that with mutated Site 1 (32.80% ± 6.89%); ( D ) In the presence of wild type Site 2, not the mutated one, miR-198 was able to significantly inhibit luciferase activity although to a less extent than wild type Site 1 (55.39% ± 8.48%). (NC, negative control; WT, wide-type; MT, mutated-type. * Compared with NC, p < 0.05).
Cyclind2, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/cyclind2/product/Boster Bio
Average 90 stars, based on 1 article reviews
cyclind2 - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

Image Search Results


MiR-198 directly bound to the 3′-UTR of CCND2 mRNA. ( A ) Bioinformatics analyses showed that 10 potential target genes of miR-198 were predicted by three different databases; ( B ) The two distinct predicted binding sites of miR-198 in the 3′-UTR of CCND2 mRNA were allocated, and the fragments containing either mutated binding site were amplified according to the mature miR-198 sequence; ( C ) In pMIR-REPORT™ vector, CCND2 mRNA 3′-UTRfragment containing either the wild type or the mutated Site 1 was fused downstream the reporter gene. When the vectors were cotransfected with miR-198 mimic or mimic control, and the relative luciferase activity, normalised by β-gal, was significantly suppressed in vector with wild type Site 1 than that with mutated Site 1 (32.80% ± 6.89%); ( D ) In the presence of wild type Site 2, not the mutated one, miR-198 was able to significantly inhibit luciferase activity although to a less extent than wild type Site 1 (55.39% ± 8.48%). (NC, negative control; WT, wide-type; MT, mutated-type. * Compared with NC, p < 0.05).

Journal: International Journal of Molecular Sciences

Article Title: miR-198 Represses the Proliferation of HaCaT Cells by Targeting Cyclin D2

doi: 10.3390/ijms160817018

Figure Lengend Snippet: MiR-198 directly bound to the 3′-UTR of CCND2 mRNA. ( A ) Bioinformatics analyses showed that 10 potential target genes of miR-198 were predicted by three different databases; ( B ) The two distinct predicted binding sites of miR-198 in the 3′-UTR of CCND2 mRNA were allocated, and the fragments containing either mutated binding site were amplified according to the mature miR-198 sequence; ( C ) In pMIR-REPORT™ vector, CCND2 mRNA 3′-UTRfragment containing either the wild type or the mutated Site 1 was fused downstream the reporter gene. When the vectors were cotransfected with miR-198 mimic or mimic control, and the relative luciferase activity, normalised by β-gal, was significantly suppressed in vector with wild type Site 1 than that with mutated Site 1 (32.80% ± 6.89%); ( D ) In the presence of wild type Site 2, not the mutated one, miR-198 was able to significantly inhibit luciferase activity although to a less extent than wild type Site 1 (55.39% ± 8.48%). (NC, negative control; WT, wide-type; MT, mutated-type. * Compared with NC, p < 0.05).

Article Snippet: The antibodies used were as follows: mouse anti-GAPDH (AG019, Beyotime, China), mouse anti-CCND2 (BA2347-2, BOSTER, Wuhan, China), Horse Radish Peroxidase (HRP)-abeled Goat Anti-Mouse Immunoglobulin G (IgG) (H + L) (A0216, Beyotime, China).

Techniques: Binding Assay, Amplification, Sequencing, Plasmid Preparation, Luciferase, Activity Assay, Negative Control

MiR-198 transfection repressed the mRNA and protein expression of CCND2. ( A ) MiR-198 mimic transfection reduced the expression of CCND2 mRNA at 24 h (68.09% ± 16.73%) and 48 h (45.68% ± 10.94%); ( B ) MiR-198 mimic or CCND2 siRNA transfection reduced the expression of CCND2 protein at 24 h (50.55% ± 24.04% or 34.45% ± 6.98%) and 48 h (17.38% ± 9.96% or 16.24% ± 9.23%). (NC, negative control. * Compared with NC, p < 0.05).

Journal: International Journal of Molecular Sciences

Article Title: miR-198 Represses the Proliferation of HaCaT Cells by Targeting Cyclin D2

doi: 10.3390/ijms160817018

Figure Lengend Snippet: MiR-198 transfection repressed the mRNA and protein expression of CCND2. ( A ) MiR-198 mimic transfection reduced the expression of CCND2 mRNA at 24 h (68.09% ± 16.73%) and 48 h (45.68% ± 10.94%); ( B ) MiR-198 mimic or CCND2 siRNA transfection reduced the expression of CCND2 protein at 24 h (50.55% ± 24.04% or 34.45% ± 6.98%) and 48 h (17.38% ± 9.96% or 16.24% ± 9.23%). (NC, negative control. * Compared with NC, p < 0.05).

Article Snippet: The antibodies used were as follows: mouse anti-GAPDH (AG019, Beyotime, China), mouse anti-CCND2 (BA2347-2, BOSTER, Wuhan, China), Horse Radish Peroxidase (HRP)-abeled Goat Anti-Mouse Immunoglobulin G (IgG) (H + L) (A0216, Beyotime, China).

Techniques: Transfection, Expressing, Negative Control

CCND2 siRNA transfection inhibited HaCaT cell proliferation by blocking cell cycle at G1 phase. ( A ) CCND2 siRNA transfection led to a significant reduction of mRNA expression of CCND2 in HaCaT cells both at 24 h (73.79% ± 11.45%) and 48 h (51.18% ± 8.85%); ( B ) Cell viability analysis showed that CCND2 siRNA transfection inhibited HaCaT cell proliferation at 24 h (67.98% ± 7.31%) and 48 h (67.45% ± 6.70%); ( C ) FCM analysis showed the effect of CCND2 siRNA transfection on cell cycle progression, and G1 phase arrest was obvious at 24 h (55.51% ± 6.18%) and 48 h (70.19% ± 4.10%) compared with negative control (42.98% ± 2.48%). (NC, negative control. * Compared with NC, p < 0.05).

Journal: International Journal of Molecular Sciences

Article Title: miR-198 Represses the Proliferation of HaCaT Cells by Targeting Cyclin D2

doi: 10.3390/ijms160817018

Figure Lengend Snippet: CCND2 siRNA transfection inhibited HaCaT cell proliferation by blocking cell cycle at G1 phase. ( A ) CCND2 siRNA transfection led to a significant reduction of mRNA expression of CCND2 in HaCaT cells both at 24 h (73.79% ± 11.45%) and 48 h (51.18% ± 8.85%); ( B ) Cell viability analysis showed that CCND2 siRNA transfection inhibited HaCaT cell proliferation at 24 h (67.98% ± 7.31%) and 48 h (67.45% ± 6.70%); ( C ) FCM analysis showed the effect of CCND2 siRNA transfection on cell cycle progression, and G1 phase arrest was obvious at 24 h (55.51% ± 6.18%) and 48 h (70.19% ± 4.10%) compared with negative control (42.98% ± 2.48%). (NC, negative control. * Compared with NC, p < 0.05).

Article Snippet: The antibodies used were as follows: mouse anti-GAPDH (AG019, Beyotime, China), mouse anti-CCND2 (BA2347-2, BOSTER, Wuhan, China), Horse Radish Peroxidase (HRP)-abeled Goat Anti-Mouse Immunoglobulin G (IgG) (H + L) (A0216, Beyotime, China).

Techniques: Transfection, Blocking Assay, Expressing, Negative Control